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While urolithin A may be able to promote better mitochondrial health by supporting mitophagy,7 there currently isn’t any research to suggest that the compound could benefit primary or genetic mitochondrial diseases (e.g., Leigh syndrome, Kearns-Sayre syndrome, Leber hereditary optic neuropathy, etc.).16 Some experts suggest that it could be worth investigating whether mitophagy activation could act as a “novel strategy” to treat these diseases, but that research hasn’t been conducted yet.17 18 19 When that happens, we’ll update this information

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This is the liver's primary detox pathway, and it is the reason liver support and glutathione are inseparable topics