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Tetrahydrobiopterin (BH 4 ) plays a vital role in this reaction by stabilizing the eNOS dimer and preventing the formation of superoxide, a potentially harmful byproduct, ensuring NO synthesis proceeds efficiently (Xia et al., 1998a
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Ferrostatin-1 is able to increase survival of UV-B-treated human lens epithelial cells and decreases the opaque region in UV-B irradiated mouse lenses [128] linking lens damage to ferroptosis
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