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Abstract Alzheimer’s disease (AD) stands as the predominant form of dementia, presenting significant and escalating global challenges
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While heterologous desensitization of the β 2 AR was mediated primarily by PKA, a novel catecholamine-activated cAMP-independent kinase, termed βAR kinase (βARK, later named GRK2), was responsible for the process of homologous desensitization [11, 12]
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