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Interestingly, several persistent clinical manifestation(s) of post-COVID symptoms in PASC patients, sustain as an aftermath from earlier SARS-CoV-2-mediated hijack of host cellular factors (i.e., ACE2, NRP1, furin, TMPRSS2, and CTSL)
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Subsequently, the toxicants progress into the distribution phase, where they exit the bloodstream and reach the target site facilitated via the porosity of the capillary endothelium, transport across the plasma, and accumulation in cellular organelles (Gupta, 2016)
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