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Macrophage studies demonstrate that GHK-Cu administration shifts these cells from a pro-inflammatory M1 phenotype to a tissue-repairing M2 phenotype, increasing their production of growth factors including vascular endothelial growth factor (VEGF) and platelet-derived growth factor (PDGF) by 40-60%
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Autocrine melatonin switches immune cells from a pro-inflammatory (M1) to an anti-inflammatory (M2) phenotype and produces a change in reactivity from cellular to humoral (Carrillo-vico et al., 2013
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