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From genetic mutations to aging, infections, and a lack of physical activity, the etiology of mitochondrial dysfunction/impairment (Figure 1) is multiple and currently an important field of research due to its implications in health and disease

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1f, 2 and Supplementary Table 1), which displays an active turnover conformation, with clear ATP-Mg 2+ density in the non-catalytic degenerate site and no detectable density in the catalytic consensus site (Fig