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The electrostatic interaction of AMPs with the bacterial membrane is imperative for their direct killing activity, occurring through physical destabilization of the membrane itself or inhibition of intracellular processes (e.g., DNA/RNA/cell wall synthesis, protein synthesis and folding, enzymatic activity, etc.) after the translocation of AMPs into the cytosol and subsequent interplay with specific intracellular targets (101), limiting the onset of resistance to AMPs due to this multitarget mechanism with complementary effects