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2017;176 doi: 10.1530/EJE-16-0927
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However, as inactivation of Cpt1b in CMs after I–R injury facilitates heart repair and restored cardiac functions—processes that cannot be explained by cardioprotection—we postulate that induction of cardiac regeneration dominates, a hypothesis that is also supported by the overall increase in CM numbers and in proliferating CMs in the infarct border zone
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