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A promising avenue involves the utilization of cysteine-rich protein-based tactics targeting cysteine-rich angiogenic inducer 61 (CCN1/CYR61) (123), which could potentially mitigate the biological aggressiveness of TNBC or BLBC
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Selenoprotein P, the main selenium transport protein in plasma (133), delivers selenium to the brain via receptor-mediated transcytosis across the blood–brain barrier (134)
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