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PRMT1 interacts with and methylates UBE2m and this interaction is enhanced in TECs with COM treatment We sought to identify potential binding proteins of PRMT1 to elucidate the mechanism of PRMT1 in regulating fatty acid metabolism
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In response to these stimuli, microglia express proinflammatory cytokines such as TNF-α, IL-1β, IL-16 and chemokines such as monocyte chemoattractant protein-1 (MCP-1) or IL-18, which are responsible for cell recruitment and removal of pathogenic substances (Glass et al., 2010
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