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parkinson glutathione oxidative stress and cellular pathologies in Parkinson's disease | Molecular Brain كريم سريع النتائج
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Indeed, we demonstrated recently in hippocampal slices that amyloid-β (Malkov et al., 2020) as well as epileptiform activity (Malkov et al., 2018) strongly inhibited glycolysis while mitochondrial function remained normal or even upregulated, potentially as a compensatory mechanism
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