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In this research, the complexity of molecular pathway systems of oxidative stress is modeled and then integrated with the recently developed in silico model of C1 metabolism in order: 1) to demonstrate the extensibility and scalability of the CytoSolve Collaboratory by coupling the in silico model of oxidative stress with the in silico model of C1 metabolism, and 2) to explore the effects of oxidative stress on C1 metabolism relative to formaldehyde (HCHO) and glutathione (GSH), a powerful antioxidant and important indicator of oxidative homeostasis in living systems

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