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To enhance therapeutic efficacy and minimize risks, future research should explore selective targeting of TNFR1 or activation of the TNF/TNFR2 pathway, which may provide a safer and more effective means of suppressing inflammation (95)

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As described previously, mTORC2 similarly regulates xCT activity, and suppresses cystine in glioblastoma, TNBC and NSCLC cell lines [22]