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Glutathione and neurodegenerative diseases: immunopharmacological implications - PMC
The ischemia triggers genetic reprogramming in efferocytes [47], resulting in their proliferation, polarization, and phagocytic enhancement through the upregulation of surface receptors, extracellular bridge molecules, cytoskeletal rearrangements, and the synthesis of anti-inflammatory and trophic factors [47, 48]
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