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Both PreC and PostC initiate the activation of these pathways during the early moments of reperfusion following the index ischemic event [57]

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Animal data demonstrates that BPC-157’s therapeutic effects continue long after administration stops: for example, in spinal cord injury studies, functional improvements and reduced spasticity were maintained for up to 360 days after a single treatment [19], while in one tendon healing study, biomechanical improvements persisted through the 21–72 day observation period [53]

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