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In a mouse silicosis model the authors reported no significant systemic toxicity
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Dosage:1mg (receptor grade)
Furthermore, subsequent addition of the complex I inhibitor rotenone entirely blocked NADH oxidation and caused a potentiation in H 2 O 2 production after mitochondrial membrane potential was collapsed by addition of FCCP (95), indicating that a high NADH level is crucial for ROS production at complex I
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