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Degradation and Metabolism of Catechin, Epigallocatechin-3-gallate (EGCG), and Related Compounds by the Intestinal Microbiota in the Pig Cecum Model
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Because cancer cells rely on constitutively elevated SMG1/NRF2 signaling because of chronic oncogenic ROS production, a condition not equivalently sustained in normal tissues, pharmacological SMG1 inhibition may preferentially compromise tumor cell redox buffering while sparing normal cells that engage this axis only transiently
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