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Quercetin also significantly improved oxidative stress by reversing H 2 O 2 -induced cell damage, decreasing ROS and apoptosis ratio, and upregulating GCLC transcription, which increased intracellular glutathione levels, ultimately alleviating oxidative stress in a caco2 cell model and DSS-induced colitis (171)
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NPC1-mTORC1 signaling couples cholesterol sensing to organelle homeostasis and is a targetable pathway in Niemann-Pick type C
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