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This transgenic mouse overexpresses the human amyloid precursor protein (APP) carrying two familial AD mutations: The Swedish mutation (KM670/671NL), which increases total Aβ production by enhancing β-secretase cleavage, and the Indiana mutation (V717F), which alters γ-secretase cleavage to favor production of the more aggregation-prone amyloid-beta 1–42 peptide (Aβ 1–42 ) [464–467]

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[DOI] [PubMed] [Google Scholar] 46.Afrin L.B., Ackerley M.B., Bluestein L.S., Brewer J.H., Brook J.B., Buchanan A.D., Cuni J.R., l Davey W.P., Dempsey T.T., Dorff S.R., et al