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Importantly, pharmacological inhibition of ferroptosis using ferrostatin‑1 or iron chelation with deferiprone has been shown to protect dopaminergic neurons and ameliorate motor deficits in toxin‑induced PD models [207], highlighting the therapeutic potential of targeting iron‑mediated toxicity

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Mitochondrial oxidative stress plays a major role in diabetic cardiomyopathy, where MnSOD overexpression has shown protective effects