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In the same study, high cholesterol also triggered ROS generation, suppressed proliferation and migration as well as inducing apoptosis and autophagy of rat TDSCs

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This can be efficiently prevented via the glyoxalase system, consisting of the highly conserved GLO1 and GLO2 enzymes that convert methylglyoxal to D‐lactate in a glutathione‐dependent manner (Sousa Silva et al ., 2013 )